Just to add some important relevant information to the answer from @Ilan.
There is no evidence for the presence of endotoxin/lipopolysaccharide in Listeria.
There was an early report of the presence of LPS (Wexler & Oppenheim, 1979) but this was contradicted by a later study (Maitra et al., 1986). The endotoxin activity of Gram negative bacteria is strongly associated with the lipid A moiety of the LPS. Lipid A is synthesised from N-acetylglucosamine in a three step pathway encoded by the genes lpxA, lpxC and lpxD. I obtained the sequences for the corresponding E. coli gene products and conducted BLAST searches against the Listeria monocytogenes genome with these results:
lpxA - best hit E=0.11
lpxC - no significant hit
lpxC - best hit E=1e-04
the last hit is to a 2,3,4,5-tetrahydropyridine-2,6-carboxylate N-succinyltransferase whereas the E. coli gene encodes a UDP-3-O-(3-hydroxymyristoyl)glucosamine N-acyltransferase. The similarity in the sequences seems to be because they are both members of a large superfamily of acyltransferases.
I conclude that Listeria monocytogenes does not not synthesise lipid A and therefore has no LPS, and no endotoxin.
Wexler, H., and J. D. Oppenheim. 1979. Isolation, characterization, and biological properties of an endotoxin-like material from the gram-positive organism Listeria monocytogenes. Infect.Immun. 23:845-857.
Maitra, SK et al. (1986) Establishment of beta-hydroxy fatty acids as chemical marker molecules for bacterial endotoxin by gas chromatography-mass spectrometry. Appl. Env. Mic. 52: 510-514