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I think it is autophagy. Lysosomal degradation. Autophagocytosis.

Example of the brown pigment (lipofuscin) here:

enter image description here

I am not sure if autophagy is the right answer to the "pathogenic" mechanism of atrophy.

Is autophagy the pathogenic mechanism of atrophy? Or what pathogenic mechanism is involved if you see lipofuscin in the histologic slide?

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Simple answer: Atrophy through autophagy.

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