(after some deliberation in the comments, I've decided to make the question more general)
An eschar or "dry scab" often forms at a site of injury over a large cut or sore.
It seems as though the healing scab portion binds more strongly to the other scab cells than to those of the surrounding healthy tissue initially, as evidenced by the ability to remove the scab en masse without disturbing the edges of the actual wound (in surgical debridement or when the area is "picked" at). Then, the eschar bonds then bonds more strongly with the edges of the wounds (so it's more difficult to remove), and then finally, the underlying newly formed skin seems to push it off completely.
Does this accurately describe the "lifecycle" of a common dry scab, or are there other well defined stages in the process? What types of cells (platelets in the beginning? epithelials at the end?) are involved with each stage of the process?