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$H_2S$ is the end product of sulfur related respirations (like sulfate respiration, sulfur respiration, etc...). By aerob (oxygen) respiration the oxygen in $O_2$ has 0 oxidation number, by $CO_2$ the oxygen has -2 oxidation number, so it was reduced while the carbon was oxidized. By the thiosulfate respiration of Salmonella enterica the following reaction ...


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SSRI's theoretically increase Serotonin levels. Serotonin is an agonist to the 5HT-3 receptors. The 5HT-3a primarily associated with the upper gut and the 5HT-3b with the lower gut. I would guess that theoretically again, SSRI's would tend to increase nausea, emesis, and diarrhea. There may be other effects, but these are the ones I have researched. The ...


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With respect to the class of specific serotonin reuptake inhibitors (SSRIs) the delayed action (say, 2 weeks) can be explained by their action on 5HT1A receptors in the dendritic region. This changes the firing pattern of the serotonergic neuron into a more fluttering mode over time, so that it releases more serotonin (5HT) Lucas et al. (2007). More 5HT ...


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The connection is one way, the input comes in the dendrites, while the output goes on the single axon in the neuron. Figure 1 - wikipedia - neuron The axon of the presynaptic neuron can connect e.g. to the dendrite of a postsynaptic neuron, and so they can form synapses in which they use neurotransmitters to transmit the signal. Figure 2 - synapse ...


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Neural axonal connections are physiologically one-way, in that an action potential would never back propagate. Since the signal is physiologically initiated near the cell body (dendritic input region) the normal flow of information therefore is dendritic-to-axonal. However, when axons are electrically activated (through neural implants such as auditory or ...


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They are two different mechanisms. Opium is arguably one of the oldest herbal medicines, being used as analgesic, sedative and antidiarrheal drug for thousands of years. These effects mirror the actions of the endogenous opioid system and are mediated by the principal μ-, κ- and δ-opioid receptors. In the gut, met-enkephalin, leu-enkephalin, β-endorphin ...



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