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Blood vessels throughout the body mostly aren't supplied by any parasympathetic fibres. But the effects of ACh through M3-ACh receptors would infact release NO (which acts on VSM and causes vasodilation), which is demonstrated after ACh intravenous administration.

My question is how is it physiologically mediated? (i.e. the ACh mediated vasodilation)

*VSM= vascular smooth muscle *NO= Nitric Oxide *Ach= Acetylcholine *M3 (Muscarinic type 3) Acetylcholine receptor

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  • $\begingroup$ ACh=acetylcholine? NO=nitric oxide? M3-ACh? Might be useful to spell out acronyms/abbreviations the first time you use them (I see you have VSM at the bottom), since odds are the average user of this site is not in your field $\endgroup$
    – Luigi
    Aug 10 at 14:26
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    $\begingroup$ Thank you! I've edited the question. $\endgroup$
    – A Nandi Ψ
    Aug 11 at 4:49

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