I have read that the hypoplasia can occur in fetal hypertrophic cardiomyocytes. However, I am interested if this can be theoretically be possible in non-fetal cardiomyocytes which are hypokinetic. I think there must be some growth factors to allow this to happen. Other proposals

  • lysozyme, sepsis and cardiac adrenergic neural response / NO / G protein 1
  • shear-wave, vortex and refolding of proteins [2 Yuan et al.]


How can hyperplasia be regulated in non-fetal hypokinesis?



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