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How would oncogenes be targeted for therapy and are there any examples of existing therapies for such cancers if the gene was upregulated (i) as a result of copy number variation and (ii) due to increased promoter activity?

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  • $\begingroup$ You mean by targeting the mutated gene in the cells or by targeting the gene product of the mutation? $\endgroup$ – Chris Apr 19 '17 at 8:58

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