Could somebody explain in simplest terms the difference between inverse agonist and neutral antagonist?
Which one is more well natural and less likely to cause pathways to rebound?
Does neutral mean less effect such that stepping from an inverse agonist to neutral is the best idea to avoid withdrawal?
Edit: I am trying to determine how quickly the pathways rebound when stopped during withdrawal. I would assume this might vary between inverse agonist and neutral antagonist. I am not asking about actual withdrawal symptoms.