Does this impulse in skeletal muscle spread much in the same way it does in neurons, with an initial potential change that spreads to its immediate surroundings and is then re-amplified or is it the case that in muscle fibers, the initially produced end plate potential must be "enough" to depolarize the whole muscle fiber?
My issue is with this bit in a lecture about NMJs I took:
Because the ACh-gated ion channels are localized to the end plate membrane, the End Plate Potential (EPP) is generated at and confined to the end plate region only and, therefore, the amplitude of EPP declines progressively with increasing distance from the end plate region.
I only found this site which talked about something similar, and there it talked about it in the context of a muscle fiber that's been blocked by a non-depolarizing neuromuscular blocker, i.e., no contraction occurs, it's just a measurement of how the EPP changes by itself, so perhaps the lecture was talking about something similar?
So, to put it simply, do electrical signals in skeletal muscles get re-amplified as they travel along the fiber, like they do with neuronal action potentials, or is the initial membrane potential sufficient and it just "spreads around"?