Why pathogenesis knowledge is increased more and more for common bacterial pathogens? Most of these studies lack a near application in disease control such as vaccines or antibiotics.

Thanks!

Examples:

Studies on the interactions of Haemophilus parasuis with porcine epithelial tracheal cells: Limited role of LOS in apoptosis and pro-inflammatory cytokine release

enter link description here

Sialic acid in the lipopolysaccharide of Haemophilus influenzae: strain distribution, influence on serum resistance and structural characterization

enter link description here

Why is knowledge of pathogenesis increased more and more for common bacterial pathogens? Most of these studies lack a near application in disease control such as vaccines or antibiotics.

Examples:

• Studies on the interactions of Haemophilus parasuis with porcine epithelial tracheal cells: Limited role of LOS in apoptosis and pro-inflammatory cytokine release

• Sialic acid in the lipopolysaccharide of Haemophilus influenzae: strain distribution, influence on serum resistance and structural characterization

Why pathogenesis knowledge is increased more and more for common bacterial pathogens? Most of these studies lack a near application in disease control such as vaccines or antibiotics.

Thanks!

Why pathogenesis knowledge is increased more and more for common bacterial pathogens? Most of these studies lack a near application in disease control such as vaccines or antibiotics.

Thanks!

Examples:

Studies on the interactions of Haemophilus parasuis with porcine epithelial tracheal cells: Limited role of LOS in apoptosis and pro-inflammatory cytokine release

enter link description here

Sialic acid in the lipopolysaccharide of Haemophilus influenzae: strain distribution, influence on serum resistance and structural characterization

enter link description here